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A Patricia Hahn, RN,MN, ARNP, replies:
I asked myself that same question several years ago, and that is when I came up with a systematic approach to ECG interpretation, the ABCDE method. The name of this method is a mnemonic for th e interpretation steps: "A" stands for Analyze rhythm; "B" for Bundle branch block; "C" for Chamber enlargement; "D" for Determine axis; and "E" for Evaluate each area for injury, ischemia, and infarction.
AAnalyze Rhythm
Assess the long lead II tracing at the bottom of the ECG. Interpret the rhythm and measure the PR, QRS, and QT intervals. A QRS duration of 0.12 seconds or greater indicates a delay in ventricular conduction. This delay could be due to a ventricular pacemaker, ventricular rhythm, or a bundle branch block. This finding is important because what depolarizes abnormally will repolarize abnormally; that is, if the QRS complex is distorted because of abnormal ventricular conduction, the ST segments and T waves will also be distorted. In step E, ST segments and T waves are assessed to determine changes related to injury (ST elevation) and ischemia (ST depression and/or T wave inversion). This finding, a wide QRS complex rhythm, will alter the interpretation of these changes on the ECG.
BBundle Branch Block
Because you have just measured the QRS complex, if the duration is within normal limits (0.040.10 seconds) then no bundle branch block is present and you can move on to the next step. However, if the QRS is equal to or greater than 0.12 seconds, there is a significant delay in conduction, possibly due to a bundle branch block. Look at V1; if the QRS has an rSR pattern, a right bundle branch block is present (Figure 1
). If the QRS complex in V1 looks wide but otherwise fairly normal, with a small R wave and a deep S wave or deep Q wave complex, a left bundle branch block is present (Figure 2
). With a bundle branch block, all of the ST and T waves will be abnormal. These changes are considered secondary ST-T wave changes and are related to the bundle branch block, rather than primary changes indicating ischemia or injury. If there is a bundle branch block on the ECG, it is difficult, if not impossible, to interpret ischemia, injury, and infarct changes when we get to step E.
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On a 12-lead ECG, it is impossible to distinguish between dilation and hypertrophy, you know only that the chamber is enlarged. Enlarged atria can be detected on an ECG with either an increased amplitude of the P wave (P-pulmonale, indicating right atrial enlargement) or an increased duration of the P wave (P-mitrale, indictating left atrial enlargement). As a reminder, normal P-wave amplitude is less than 2.5 mm in height, and normal P-wave duration is <0.11 seconds. Assess lead II, because it tends to best reflect atrial activity.
P-pulmonale, right atrial enlargement, is noted by a tall, peaked P wave with an amplitude exceeding 2.5 mm. P-mitrale, left atrial enlargement, will develop a notched or M-shaped P wave, with a duration that exceeds 0.11 seconds.
Enlargement of the ventricles is assessed by looking at the precordial leads V1 through V6. V1 and V2 tend to be the more right sided of the precordial leads, and V5 and V6 tend to be more left sided. When a chamber is enlarged, it will produce more electrical current. This change is detected on an ECG as increased amplitude, or positive forces recorded as R waves on the ECG. If the right ventricle is enlarged, tall R waves will be detected in V1 and V2, and deep S waves (negative forces) in V5 and V6. Right ventricular enlargement is rarely detected on an ECG, because the left ventricle is normally 2 to 3 times the muscle mass compared with the right ventricle. There are 3 other ECG findings that may also result in tall R waves in V1 and V2, but they will not cause deep S waves in V5 and V6; these findings are right bundle branch block, Wolff-Parkinson-White syndrome, and a posterior wall infarction.
Left ventricular enlargement is a fairly common finding on an ECG. It is detected by tall R waves in V5 and V6, with deep S waves in V1 and V2. Ventricular enlargement will often cause an axis deviation, because the axis tends to shift towards areas of hypertrophy.
DDetermine Axis
The mean QRS axis simply refers to the average direction or flow of current as it depolarizes the myocardium. Generally, current travels down and to the left; therefore, a normal axis would be in the left lower quadrant, 0 to +90° (Figure 3
). If the axis deviates too far to the right, it would be in the +90° to ±180° quadrant, indicating a right axis deviation; if it deviates too far to the left, (0 to 90°), a left axis deviation is present; and if it falls in the 90° to ±180° quadrant, it is considered an indeterminate axis. Normal axis is extended up to the 30° range, to account for the natural shift of the heart to the left. So, technically 30° to +90° is considered a normal axis.
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Injury, an acute, yet reversible process, indicates impending myocardial damage. It is detected by ST-segment elevation of 1 mm or more in the limb leads, and 2 mm or more in the precordial leads. Ischemia, also a reversible process, is seen as ST-segment depression and/or T-wave inversion. Infarction indicates permanent myocardial damage. In the limb leads, Q waves will form as a sign of an infarct. A significant Q wave, indicating infarction, must meet 3 criteria:
In the precordial leads (V1 through V6), infarction will be detected by a loss of R waves. Normally R waves progress, becoming larger as you move from V1 to V4, then becoming slightly smaller in leads V5 and V6. Loss of R waves indicates a loss of positive forces because of an infarction.
Myocardial infarctions are described according to the wall of the left ventricle affected. The left ventricle essentially has 5 areas or walls, the anterior, septal, lateral, inferior, and posterior walls. The ECG leads are placed in different positions on the chest and limbs, and look at the various walls of the left ventricle. Leads II, III, and aVF reflect changes in the inferior wall. V1 through V4 assess the anterior and septal walls. Lead I, aVL, V5, and V6 reflect the lateral wall. The posterior wall may be assessed directly with extended precordial leads V7, V8, and V9 or through reciprocal changes in V1 and V2. Right ventricular infarcts often occur with inferior wall infarcts, and occlusion of the right coronary artery. To detect a right ventricular infarct, right-sided precordial leads may be obtained. V4R is the most accurate lead to detect right ventricular infarcts.
This simplified ECG interpretation method, divided into 5 easy steps, covers the basics of 12-lead ECG interpretation, and is not meant to be a comprehensive interpretation. As with any skill, a little practice goes a long way.
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